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Dentinogénese imperfeita: características clínicas, diagnóstico e plano de tratamento - uma revisão narrativa da literatura
| Name: | Description: | Size: | Format: | |
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| PPG_35155 | 1.07 MB | Adobe PDF |
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Abstract(s)
A dentinogénese imperfeita é uma anomalia da estrutura dentária hereditária que é transmitida
de forma autossômica dominante. Esta patologia afeta um número estimado de 1 em cada 8000
pessoas. De acordo com a classificação de Shield, pode ser dividida em 3 tipos. A dentinogénese
imperfeita tipo 1 está associada à osteogénese imperfeita. A Dentinogénese imperfeita tipo 2,
é a mais frequente e não há envolvimento ósseo. O esmalte tem uma modificação na junção
dentina esmalte, com hipomineralização o que leva à sua perda precoce, e exposição dentinária
que rapidamente sofre atrição. As raízes são curtas e estreitas com possível obliteração pulpar.
A Dentinogénese imperfeita tipo 3 é encontrada numa população isolada em Brandywine,
Maryland.
É desejável uma abordagem o mais precoce possível, a fim de interceptar a atrição e preservar
a função, a estética e o crescimento normal para manter uma relação inter-arcada adequada.
Pretendeu-se com esta dissertação efectuar uma revisão da informação publicada sobre esta
anomalia e a sua forma de abordagem e para tal foi efectuada uma pesquisa nas bases
Pubmed/Medline e Scielo, a fim de reunir a literatura com maior evidência científica.
Dentinogenesis imperfecta is an anomaly of the hereditary dental structure that is transmitted in an autosomal dominant way. This condition affects an estimated number of 1 in 8000 people. This disease is divided into 3 types according to the classification of Shield in Dentinogenesis imperfecta type 1 associated with osteogenesis imperfecta. Dentinogenesis imperfecta type 2, is the most frequent and there is no bone involvement. The enamel has a modification of the dentin junction enamel, with hypomineralization which leads to its early loss, and dentinal exposure that quickly undergoes attrition. The roots are short and narrow with possible pulp obliteration. Dentinogenesis imperfecta type 3 is found in an isolated population in Brandywine,-Maryland. An approach as early as possible is desirable in order to intercept attrition and preserve function, aesthetics and normal growth in order to maintain an adequate inter-arcade relationship. The purpose of this dissertation was to carry out a review of the published information on this anomaly and its form of approach, and a search was carried out on the basis of Pubmed/Medline and Scielo, in order to gather literature with greater scientific evidence.
Dentinogenesis imperfecta is an anomaly of the hereditary dental structure that is transmitted in an autosomal dominant way. This condition affects an estimated number of 1 in 8000 people. This disease is divided into 3 types according to the classification of Shield in Dentinogenesis imperfecta type 1 associated with osteogenesis imperfecta. Dentinogenesis imperfecta type 2, is the most frequent and there is no bone involvement. The enamel has a modification of the dentin junction enamel, with hypomineralization which leads to its early loss, and dentinal exposure that quickly undergoes attrition. The roots are short and narrow with possible pulp obliteration. Dentinogenesis imperfecta type 3 is found in an isolated population in Brandywine,-Maryland. An approach as early as possible is desirable in order to intercept attrition and preserve function, aesthetics and normal growth in order to maintain an adequate inter-arcade relationship. The purpose of this dissertation was to carry out a review of the published information on this anomaly and its form of approach, and a search was carried out on the basis of Pubmed/Medline and Scielo, in order to gather literature with greater scientific evidence.
Description
Keywords
Dentinogénese imperfeita Ostéogénese imperfeita Displasia dentinária Dentina opalescente Anomalias dentinárias Tratamento da DGI Dentinogenesis imperfecta Osteogenesis imperfecta Dentin dysplasia Dental anomalies Opalescent dentin Dentinal anormalities Treatment of DGI